What are the basic understandings of the brain, the mind, and the soul of near-death experiences? In the first of three volumes, Christianity and the Brain, Volume 1: Faith and Medicine in Neuroscience Care delves into the fascinating aspects of the human brain?God?s hidden treasure?and its development. Inspired by the Coptic Orthodox faith while growing up in Egypt, Christian neurological surgeon and anesthesiologist Ramsis F. Ghaly uses his experiences to reflect on spirituality and science and the ties between Christianity and the human brain. He also explores neuroscience and God, faith and medicine, the universe and heaven, and birth and life beyond death. Through Ghaly?s innovative research, you will grow closer to the Creator and learn to understand Him like never before. A medical career is a sacred vocation with high ethical morals and values. In accordance with such standards, Ghaly illustrates the ideal neuroscience health-care structure in view of holism and patient empowerment, especially toward the dire need of modern care in the world, including the United States of America. Powerful and informative, Christianity and the Brain, Volume 1 takes a new perspective on a seldom-studied subject.
This digital document is an article from Journal of Neuroscience Nursing, published by American Association of Neuroscience Nurses on February 1, 1998. The length of the article is 5735 words. The page length shown above is based on a typical 300-word page. The article is delivered in HTML format and is available in your Amazon.com Digital Locker immediately after purchase. You can view it with any web browser.From the supplier: A prospective study exploring the effect of reduced spasticity for quality of life (QOL) has been carried out using intrathecal baclofen. Spasticity was greatly reduced in 25 patients treated for one year. QOL was not shown to change statistically based on the Ferrans and Powers QLI. On the health and functioning subscale there was a trend toward improvement.Citation DetailsTitle: Quality of life: effect of reduced spasticity from intrathecal baclofen.Author: Janet M. GianinoPublication: Journal of Neuroscience Nursing (Refereed)Date: February 1, 1998Publisher: American Association of Neuroscience NursesVolume: v30 Issue: n1 Page: p47(8)Distributed by Thomson Gale
This digital document is a journal article from Neurobiology of Learning and Memory, published by Elsevier in 2007. The article is delivered in HTML format and is available in your Amazon.com Media Library immediately after purchase. You can view it with any web browser.Description: Experiences with a high emotional content (aversive) tend to be stored as long-term memories; however, there are also contextual recollections, which form a significant part of our memories. Different research has shown that the insular cortex (IC) plays an important role during aversive memory formation, yet its role during incidental/non-aversive learning like pre-exposure contextual memory formation has received little attention. The objective of this research was to establish the role of cholinergic activity in the IC through its muscarinic receptors during the formation of inhibitory avoidance (IA) memory, as well as during pre-exposure contextual memory, using a paradigm such as latent inhibition (LI). Rats with bilateral cannulae directed into the IC were trained in the LI paradigm of IA or IA task alone. The muscarinic antagonist receptor scopolamine was infused bilaterally into the IC 5min before the pre-exposure into the dark chamber of the IA cage, one day before the conventional IA training or during the IA training day. During the IA test, the entrance latency into the dark chamber of the IA cage was measured as an index of contextual memory. The results showed that scopolamine infused before and after IA training disrupts inhibitory avoidance memory. Also, it showed that the pre-exposed saline-infused animals (LI) had a lower entrance latency compared to the group not pre-exposed (IA). However, the group that received scopolamine into the IC before, but not after, the pre-exposure to the dark chamber, presented a similar latency to the IA group, showing a blockade of the latent inhibition of the IA. These results suggest that cholinergic activity in the insular cortex is necessary during the acquisition and consolidation of avoidance memory, but appears necessary only during the acquisition of pre-exposure non-aversive contextual memory.
This digital document is an article from Journal of Neuroscience Nursing, published by Thomson Gale on December 1, 2007. The length of the article is 5160 words. The page length shown above is based on a typical 300-word page. The article is delivered in HTML format and is available in your Amazon.com Digital Locker immediately after purchase. You can view it with any web browser.Citation DetailsTitle: Quality of life and burden in caregivers of patients with epilepsy.(Clinical report)Author: Ana Carolina Westphal-GuittiPublication: Journal of Neuroscience Nursing (Magazine/Journal)Date: December 1, 2007Publisher: Thomson GaleVolume: 39 Issue: 6 Page: 354(7)Article Type: Clinical reportDistributed by Thomson Gale
This digital document is a journal article from Neurobiology of Learning and Memory, published by Elsevier in . The article is delivered in HTML format and is available in your Amazon.com Media Library immediately after purchase. You can view it with any web browser.Description: Human episodic memory refers to the recollection of an unique past experience in terms of its details, its locale, and temporal occurrence. Episodic memory, even in principle, has been difficult to demonstrate in non-verbal mammals. Previously, we provided evidence that mice are able to form an integrated memory for ''what,'' ''where,'' and ''when'' aspects of single experiences by combining different versions of the novelty-preference paradigm, i.e., object recognition memory, the memory for locations in which objects were explored, and the temporal order memory for objects presented at distinct time points. In the present series of experiments we evaluated whether this paradigm, with minor modifications, also works with rats. We found that rats spent more time exploring an ''old familiar'' object relative to a ''recent familiar'' object, suggesting that they recognized objects previously explored during separate trials and remembered their order of presentation. Concurrently, the rats responded differentially to spatial object displacement dependent on whether an ''old familiar'' or ''recent familiar'' object was shifted to a location, where it was not encountered previously. These results provide strong evidence that the rats established an integrated memory for ''what,'' ''where,'' and ''when.'' We also found that acute stress impaired the animal's performance in the episodic-like memory task, which, however, could be partially reversed by the N-Methyl-d-aspartate-receptors agonist d-cycloserine.
This digital document is a journal article from Neurobiology of Learning and Memory, published by Elsevier in 2005. The article is delivered in HTML format and is available in your Amazon.com Media Library immediately after purchase. You can view it with any web browser.Description: Unilateral entorhinal lesions have enjoyed immense popularity as a model of recovery from damage. In part, the popularity has been supported the laminar organization of the hippocampal formation, which allows for the dissection of the contribution of individual afferent pathways to the recovery process. The commissural/associational pathway is of particular interest, since electrophysiological and gross anatomical data, although limited, have correlated sprouting in this pathway with behavioral recovery. Unfortunately, information relating recovery to synaptic structure is lacking. Addressing this issue, two analyses were conducted. Initially, a quantitative review of the literature reporting behavioral recovery following this type of lesion was conducted using meta-analytic techniques. Using this detailed information across decades of research, multiple linear regression analysis was conducted to address whether the morphological correlates of recovery could predict behavioral recovery. This resulted in an equation relating morphology and recovery that stood up well to several diagnostic tests. Moreover, this model suggests that synapse structure (in particular, synapse size and curvature, as well as terminal compartmentalization and the density of multi-synaptic terminals) holds a greater potential to predict behavioral recovery than increases in synapse number, which is typically seen as the optimal anatomical measure of recovery. This initial attempt to identify, quantify, and validate a model of lesion recovery is an important initial step in understanding how synaptic morphology may help mediate recovery of function.